If air conduction is also poor, along with the malfunctioning of the outer and middle ear, conductive hearing loss starts to develop (Schwartz, 1996). Genetic hearing loss is caused by the mutations in human genes, and can sometimes be prevented and known to patients due to their family history (Schwartz, 1996). A baby can be exposed to hearing loss if its family has histories of hearing loss, maternal illnesses, and even if a baby is born under 3.3 pounds (Schwartz, 1996). Autosomal recessive conditions are inherited mutated genes from parents which causes genetic hearing loss, as well (Schwartz, 1996). Nongenetic hearing loss can be caused by unusual and premature ear structures (Schwartz, 1996). Maybe a sign of undiscovered hearing loss is a person’s slurred and improper speech (Schwartz, 1996). When someone has mixed hearing loss, he or she has sensorineural hearing loss and conductive hearing loss (Schwartz, 1996). When spiral ganglion neurons stop sending nerve impulses from the cochlea to the brain, and there is a loss of sensory hair cells in the ear, deafness occurs (Chen & et al., 2012). About four-thousand newly born babies in the United States of America are born deaf (Schwartz, 1996). Ten percent through ninety percent of the usual number of auditory nerve fibers are present in deaf children (Schwartz, 1996). Two percent …show more content…
Tinnitus is a high-pitched sound that is related to hearing loss and includes all the sounds heard without an eternal sound equivalent (Frosch, 2013). Small microphones in the ear canal can measure spontaneous otoacoustic emissions (SOAE’s), which are noises radiated from the inner ear and cochlea (Frosch, 2013). Spontaneous otoacoustic emissions are given off by the moving characteristics of the outer ear hair cells and instinctive mechanical oscillations found inside the cochlea (Frosch, 2013). These emissions are quite a fascinating and captivating subject for research (Frosch, 2013). Also, SOAE’s can be disclosed in half of the population (Frosch, 2013). Unusual auditory brain response (ABR), absent normal otoacoustic emissions (OAE), and severely inflated middle ear muscle reflexes are components of AuditoryNeuropathy/Dysynchrony (AN/AD) (Schwartz, 1996). David Kemp was a famous scientist who discovered spontaneous otoacoustic emissions in 1978 (Frosch, 2013). His discovery brought attention to this area of the auditory field (Frosch,