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35 Cards in this Set
- Front
- Back
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Analgesia
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Freedom from or absence of pain
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Tranquilization
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Behavioral change wherein anxiety is relieved and the patient becomes relaxed but is aware of its surroundings
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Sedation
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State characterized by central depression accompanied by drowsiness
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Narcosis
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Drug induced state of deep sleep from which the patient cannot easily be aroused
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Hypnosis
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Artificially induced sleep or trance resembling sleep
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Local Anesthesia
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Loss of sensation in a circumscribed area of the body
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Regional Anesthesia
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Insensibility in a larger, though limited area of the body
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General Anesthesia
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Drug induced unconsciousness that is characterized by controlled but reversible depression of the CNS
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Surgical Anesthesia
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State or plane of general anesthesia that provides UNCONSCIOUSNESS, MUSCULAR RELAXATION AND ANALGESIA required for painless Sx
"The Triad" |
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Balanced Anesthesia
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The use of multiple drugs from different classes to specifically attenuate individual components of the anesthetic state
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Dissociative Anesthetics
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Dissociation of the thalamocortic and limbic systems characterized by a cataleptoid state in which the eyes remain open and the swallowing reflex remains intact.
Commonly seen with Ketamine |
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What is the overall/general anesthesia plan?
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Formulate a Plan
Premedicate Induction Maintenance Monitoring Supportive Care Recovery |
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What are the 5 categories we place patients in prior to Sx?
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I - Normal Healthy Patient
II - Patient has mild or local Systemic Dz III - Patient has severe systemic dz IV - Patient has severe systemic dz that is a constant threat to life V - Moribound patients not expected to live 1 day with or without Sx |
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What are the BIGGEST anesthetic complications and concerns?
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Hypothermia
~ Most common complication Hypotension ~ Most anesthetics cause vasodilation ~ Inhalents are very potent vasodilators Hypoventilation ~ Common secondary to anesthetic and analgesic drugs Bradycardia ~ Secondary to increased vagal tone from drugs such as Opiods, Alpha-2-agonists, and inhalents Pain |
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Purpose of premedications and some examples.
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Reduce anxiety and stress
Provides analgesia Reverses unwanted side effects Reduces induction and inhalant requirements Alpha-2-agonists, Opiods, Benzodiazepines, Phenothiazines, Anticholinergics, Dissociatives |
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What to do after premedicating patient.
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Wait 15-20 minutes for drugs to take effect
Place IV cath Prepare for induction ~ Prepare induction agent ~ IV flush solution (Hep/saline) ~ Endotrach tubes, Laryngescope, gauze ties, lube ~ IV fluids ~ Pre-Oxygenate patient!! (about 10 min) especially since we are using propofol |
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How to induce.
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Prior to induction
~ Thoracic auscultation and determine HR Administer Induction Agent ~ In Jx its propofol, SLOWLY Determine if depth is adequate for intubation |
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What do induction agents do and what are some examples?
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Produce smooth induction with rapid progression through excitement stage (Stages I and II)
Allows for rapid control of airway Propofol, Thiopental, Ketamine, Etomidate, High dose Opiods, Inhalants |
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MAC for:
Isoflurane Sevoflurane |
Iso - 1.3
Sevo - 2.3 |
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Surgical MAC for:
Isoflurane Sevoflurane |
MAC x 1.5
Iso - 2% Sevo - 3.45% |
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How to increase alveolar concentration. The three alveolar inflow factors...
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Vaporizer Setting
Fresh Gas flow rate ~Set flow meter to 2 L/min Breaths per minute |
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Stage I of Anesthesia
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Starts as soon as syringe enters cathater
Stage of coluntary movement Lasting from initial administration of loss of conciousness |
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Stage II of Anesthesia
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Stage of delirium or involuntary movement
Lasting from loss of consciousness to the onset of a regular pattern of breathing |
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Stage III of Anesthesia
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Stage of SURGICAL ANESTHESIA
Further divided into planes 1 to 4 Stage III, plane 2 provides adequate muscle relaxation and analgesia for most surgical procedures Plane I (light anesthesia) Plane II (medium anesthesia) Plane III Plane IV (deep anesthesia) |
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Stage IV of Anesthesia
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Extremely depressed CNS
Respiratory Ceases Heart beats for only a short time Cardiovascular collapse and shock Death occurs quickly unless CPCR is begun |
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How to monitor patients under anesthesia
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Physical Signs
~ Jaw tone ~ Palpebral reflex ~ Corneal reflex ~ Normals (HR, RR, Systolic mean and diastolic BPs) Equipment ~ ECG ~ BP ~ SPO2 ~ ERCO2 |
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Supportive care during anesthesia
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IV Crystaloid Fluids
~ Lac Ringers, Normasol or 0.9% NaCl ~ 10ml/kg/hr then convert to drops/sec ~ Helps w/ vasodilation caused by anesthesia. The fluids will help inc BP Supplemntal heat |
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Stage III of Anesthesia
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Stage of SURGICAL ANESTHESIA
Further divided into planes 1 to 4 Stage III, plane 2 provides adequate muscle relaxation and analgesia for most surgical procedures Plane I (light anesthesia) Plane II (medium anesthesia) Plane III Plane IV (deep anesthesia) |
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Stage IV of Anesthesia
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Extremely depressed CNS
Respiratory Ceases Heart beats for only a short time Cardiovascular collapse and shock Death occurs quickly unless CPCR is begun |
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How to monitor patients under anesthesia
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Physical Signs
~ Jaw tone ~ Palpebral reflex ~ Corneal reflex ~ Normals (HR, RR, Systolic mean and diastolic BPs) Equipment ~ ECG ~ BP ~ SPO2 ~ ERCO2 |
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Supportive care during anesthesia
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IV Crystaloid Fluids
~ Lac Ringers, Normasol or 0.9% NaCl ~ 10ml/kg/hr then convert to drops/sec ~ Helps w/ vasodilation caused by anesthesia. The fluids will help inc BP Supplemntal heat |
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Reflex behavior at proper anesthetic depth
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Palpebral: dogs may have no reflex at adequate surgical depth
Corneal: should always be present Nystagmus: Excitement or light Lacrimation: parasympathetic stim, light plane Medioventral eye position: most desirable, central is too deep Jaw tone: moderate to loose |
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Most reliable signs of anesthetic depth
Also somewhat reliable signs of depth |
Most:
Gross purposeful movement Reflex movement Immediate hemodynamic response Immediate resp response Somewhat: Hx of vaporizer setting (MAC) Muscle tone Pupillary, Palpebral, Corneal reflexes Moist Cornea Eye Position |
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Less reliable signs of anesthetic depth
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HR
RR BP |
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The most effective anesthetic depth is on that....
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Obliterates the animals response to noxious stimuli without depressing circulatory and respiratory systems
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