- Shuffle
Toggle OnToggle Off
- Alphabetize
Toggle OnToggle Off
- Front First
Toggle OnToggle Off
- Both Sides
Toggle OnToggle Off
Front
How to study your flashcards.
Right/Left arrow keys: Navigate between flashcards.right arrow keyleft arrow key
Up/Down arrow keys: Flip the card between the front and back.down keyup key
H key: Show hint (3rd side).h key
![]()
PLAY BUTTON
![]()
PLAY BUTTON
![]()
113 Cards in this Set
- Front
- Back
|
True or false. Birds do not have sweat glands.
|
True
|
|
Compare the skin of birds to that of mammals.
|
Thin (except for "scaled" areas)
|
|
Why is molting so stressful?
|
Because it takes lots of proteins to make feathers so can be opportunity for disease, but must molt yearly
|
|
What do they think may be a use for the uropygial glands (greasy glands on butt)?
|
Maybe communication
|
|
What is different about the vertebral column of bird?
|
T2-T5 and T6/T7-L7 fused
-Articulation b/w T5 & T6 -Cervical vertabrae varies from 11-25 |
|
Except for the ____ and ______ there are no bony epiphyses in birds.
|
Tibia and proximal metatarsus
|
|
Many avian bones are "aerated" and provide an important mineral reserve for _____.
|
Egg production
|
|
The _____ joint is a major stress point for birds; the ______ bone is the fastest growing bone in the bird's body.
|
Intertarsal joint=major stress point
Tibiotarsal bone=fastest growing |
|
How many occipital condyles do birds have?
|
one
|
|
How does the skeleton of ratite birds vary from carinate birds?
|
Ratite: have no keel and are ground birds
Carinate: have a keel and can fly |
|
Birds have _____ girdles.
|
Pelvic girdles
|
|
What is the content of "red" muscle in birds?
|
High fat, low glycogen
|
|
Birds have oxygen exchange during ______.
|
Both inspiration and expiration; countercurrent exchange
|
|
Pathologies of the avian respiratory system tend to be located _____.
|
Dorsoposterior
-Gravity? |
|
What are the clinical signs of respiratory issues in birds?
|
Snick, sneeze, gurgle and increased lacrimation
-No coughs w/ birds |
|
Some birds have extensive ______ sinuses.
|
Cervicocephalic sinuses
|
|
What are the 5 locations of avian air sacs?
|
Anterior, posterior and thoracic air sacs, abdominal, interclavicular air sac
|
|
The heart of birds tends to be _____ shaped.
|
Cone shaped
|
|
What is different about the right AV valve in birds?
|
It's a muscular flap
|
|
Minimal _____ lumen is seen on the heart of birds at necropsy.
|
Left ventricular lumen
|
|
The renal portal system sends blood from hind limbs through the kidneys, but what can happen during stress?
|
This blood can be shunted directly to caudal vena cava or to the hepatic portal vein
|
|
The BP of birds is _____ than most animals.
|
Higher (200 mm Hg in turkeys)
|
|
Birds lack lymph nodes, so what do they have instead?
|
Lymphoid follicular aggregates in various tissues, including Harderian glands (eyelid) and intestinal wall, cecal tonsils
|
|
What is different about the spleen in birds?
|
Lymphoid rich
-Various shapes-can be round |
|
**When does the cloacal bursa begin to involute in birds?
|
14 weeks (complete atrophy by 22 weeks)
|
|
Explain the development of the avian thymus with time.
|
Enlarges until sexual maturity (18 weeks) then atrophies
|
|
What are polymorphonuclear cells called in birds?
|
Heterophils (not neutrophils)
|
|
Thrombocytes are _____, nucleated and the same size as lymphocytes in birds.
|
Phagocytic
|
|
How does the avian urinary system eliminate waste?
|
Uric acid metabolism
-Drains into cloaca where it can mix w/ feces |
|
The ______ is a common site for extramedullary hematopoiesis in birds.
|
Kidney
|
|
What is different about the anatomy of the avian kidney?
|
No clear gross distinction b/w medulla & cortex
|
|
What structure is completely missing in the avian urinary system?
|
No urinary bladder
|
|
What is different about the avian central nervous system?
|
Well developed cerebellum
-Poor cerebral development -Ocular specializations |
|
For the most part, what do avian salivary glands secrete?
|
Only mucus
|
|
Crops are not present in _____ and some fish-eating birds.
|
Owl
|
|
What are the 2 parts of the avian stomach?
|
Proventriculus (secretory) before ventriculus (grinding koilin surface)
|
|
What is different about the avian small intestine compared to mammals?
|
Generally shorter than mammals
|
|
Bird droppings contain both ____ and ____ component.
|
Renal and GI components
|
|
The organization of the avian liver lacks the clear-cut ______ of mammals.
|
Acinar pattern
|
|
What is different about the gene makeup of male and female birds?
|
Females are heterogametic: ZW
Males are homogametic: ZZ |
|
In female birds abiotrophy of the _____ side occurs in the reproductive system.
|
Right side
|
|
What are the 5 parts of the avian oviduct?
|
1) Infundibulum
2) Magnum 3) Isthmus 4) Shell gland (uterus) 5) Vagina |
|
What is the urodeum segment of the cloaca in birds?
|
The urodeum is the smallest cloacal chamber in psittacines. It receives the ureters, the oviduct (females), and ductus deferens (males)
|
|
How often can chickens lay eggs?
|
Every 24 hours
|
|
Compare the basal metabolic rate of birds to mammals.
|
Birds have a higher basal metabolic rate with temp around 40-42 degrees C
|
|
True or false. It takes weeks for the mass of the avian pectoral muscle to decrease.
|
False, takes just a few days- higher basal metabolic rate than mammals
|
|
Birds need feathers to regulate _____.
|
Heat
|
|
Birds don't have a peritoneal/thoracic cavity, they have ______.
|
Coelom-actually a total of 16 cavities (8 are air sacs)
|
|
Is avian or mammalian inflammation more rapid?
|
Avian is much more rapid
|
|
What type of inflammatory reaction do birds have?
|
Intense granulomatous reaction (within 12 hours)
|
|
_______ are involved in the acute inflammatory response but not eosinophils.
|
Basophils
|
|
Are macrophages or heterophils more active phagocytes?
|
Macrophages
|
|
Avian ______ and _____ have defective lysosomal activity.
|
Heterophils
Thrombocytes |
|
What is different about the lymphoid reaction in birds?
|
It quickly dominates, resulting in lymphofollicular nodules at site of insult
-Not good at making liquid exudate |
|
What occurs b/w 1-6 hours of the avian inflammatory response?
|
Influx of basophils, heterophils and monocytes
|
|
What occurs b/w 6-12 hours of the avian inflammatory response?
|
Lymphocytes predominate + monocyte/macrophage
|
|
What occurs b/w 12-36 hours of the avian inflammatory response?
|
Giant cells start to appear
-Peak by 36 hours. -Heterophils persist if stimulus remains |
|
What happens during 36-72 hours of the avian inflammatory response?
|
-Regeneration and repair (fibroblasts)
-Secondary lymphoid follicles -**Caseation and granuloma fomation |
|
True or false. Birds do generate fibrin.
|
True
|
|
A bird presents to you with small tissue growths in the apteric areas such as around the eyes and mouth. What's high on your differentials? How can you diagnose it?
|
Poxvirus!
Looks for inclusion bodies on a slide |
|
What causes fowl cholera?
|
=Avian pasteurellosis (pasteurella multocida)
|
|
All poultry are susceptible to fowl cholera, but is a key disease in _______.
|
Turkeys
|
|
The clinical signs of fowl cholera are _______ related.
|
Respiratory related
|
|
What will you see on necropsy of a bird with fowl cholera?
|
Widespread hemorrhage and necrosis
|
|
Why does fowl cholera cause arthritis?
|
Because fowl cholera causes a strong inflammatory response and chronic infections can localize (arthritis, tendons, ears, etc)
|
|
What's the treatment for fowl cholera?
|
Antibiotics, attenuated vaccines
|
|
What is ILT?
|
Infectious laryngotracheitis
-Highly contagious respiratory disease |
|
ILT mainly affects what bird?
|
Chickens
|
|
What is the mortality of ILT in naive flocks?
|
Can be over 50%
|
|
What can be done to prevent infectious laryngotracheitis?
|
Vaccine available
|
|
What are 3 clinical signs of ILT?
|
Gasping, coughing (bad news), extension of neck
|
|
What kind of virus is the infectious laryngotracheitis virus?
|
A herpesvirus
|
|
What can you see on necropsy of a chicken with ILT?
|
May see yellow nodules in respiratory system
|
|
What is a fungus that affects birds?
|
Aspergillosis
|
|
What causes infectious bursal disease?
|
A birnavirus
|
|
What form of infectious bursal disease is the most serious and why?
|
Subclinical form is most serious bc it leads to "disaster flocks"
|
|
When are chickens usually infected w/ the birnavirus that causes infectious bursal disease?
|
Chicks usually infected before 3 weeks of age
-Bursa normally ceases to grow by about 14 weeks and atrophies by time of sexual maturity |
|
What are 2 types of lymphoid neoplasia in chickens?
|
1) Marek's disease
2) Lymphoid leukosis |
|
At what age does Marek's disease usually infect chickens? What causes Marek's disease?
|
It's a herpes virus, often infects 3-30 weeks of age
|
|
What type of a tumor is produced by Marek's disease? Lymphoid leukosis?
|
Marek's=T cell tumor
Lymphoid leukosis=B cell tumor |
|
Does marek's or lymphoid leukosis cause paralysis?
|
Marek's disease may
Lymphoid leukosis does not |
|
Is marek's disease or lymphoid leukosis not neurotropic?
|
Lymphoid leukosis is not
Marek's disease often is neurotropic |
|
How does Marek's disease affect the bursa? Lymphoid leukosis?
|
Marek's disease: bursa enlarged or atrophied
Lymphoid leukosis: may see bursal tumors |
|
Does Marek's disease or lymphoid leukosis have a vaccine available?
|
Marek's=vaccine
Lymphoid leukosis=no vaccine |
|
Marek's or lymphoid leukosis.... infiltrates diffusely.
|
Lymphoid leukosis
|
|
What are 2 things you might see upon necropsy of a bird with Marek's virus?
|
Big sciatic nerve
Tiny nodules that are foci of follicular replication |
|
Birds get host-specific and even site specific infections by _____.
|
Eimeria spp
|
|
What are 2 examples of Eimeria species that infect chickens?
|
E. tenella (ceca)
E. necatrix (anterior SI) |
|
What is the prepatent period of eimeria in birds? Where do the oocysts sporulate?
|
4-7 d
Sporulate in environment and are generally quite hardy |
|
What are the clinical signs of coccidiosis in birds?
|
Varies from poor weight gain to unanticipated death
|
|
If a bird is raised on solid ground how should you treat coccidiosis?
|
Manage with anticoccidial drugs +/-vaccination
-Coccidiostatic vs coccidiocidal drugs |
|
"________programs" are used to treat coccidiosis in order to decrease drug resistance.
|
Shuttle programs -change drugs a lot
|
|
Where are birds most likely to get dyschondrodysplasia?
|
Tibia..
|
|
What will you see on necropsy in a bird with Heterakis sp.? How is Heterakis spread?
|
Hepatitis
Tiphlitis Black head -Spread by roundworms |
|
What is a common problem in laying hens?
|
Hepatic lipidosis
|
|
What are the 2 most important causes of mortality in baby chicks?
|
1) Starvation-they're dumb
2) Yolk ac infection |
|
What will you see on necropsy of a bird with ascites syndrome?
|
Coelom filled w/ fibrin and fluid
-Genetic problem |
|
Other than hematopoietic neoplasias, most neoplastic problems in birds are found in ___________.
|
Elderly laying hens
|
|
What must you rule out before diagnosing "carcinamatosis" due to ovary/oviduct, pancreas or intestinal neoplasia?
|
Avian tuberculosis
|
|
_________ (neoplasia) of the oviduct, and ___________ are also common neoplasms of birds.
|
Leiomyomas of oviduct
Squamous cell carcinomas |
|
What causes "fowl plague"?
|
Avian influenza
|
|
What kind of virus is avian influenza? How are the variants classified?
|
Orthomyxoviridae, type A strain
Classified via combination or HA and N antigens |
|
There's variability in the virulence of mammalian influenza because the and viruses are constantly mutating via _______.
|
Antigenic drift/shift
|
|
*What is the reservoir for avian influenza?
|
Water fowl
|
|
Avian influenza can infect what other animal?
|
Felids
|
|
True or false. Avian influenza cannot survive in the environment.
|
False, hardy in environment and cool temperatures
|
|
Why is avian influenza tracked so closely when there are other diseases with a higher mortality?
|
Has the potential to spread very rapidly
*Can infect humans |
|
**What are the 7 clinical signs of avian influenza?
|
1) Depression, inappetence
2) Drop in egg production 3) Spike in mortality rates 4) Respiratory distress 5) Edema, cyanosis of face, comb, wattles 6) Hemorrhages 7) Incoordination |
|
What are some gross lesions associated with avian influenza?
|
-Hemorrhage
-Cyanotic comb -Target lesions in pancreas from necrosis |
|
What are 3 things to look for to diagnose Avian influenza?
|
Hemorrhage, high degree of mortality, evidence of respiratory distress
|
|
What are 3 differentials that cause similar signs to avian influenza (hemorrhage, respiratory signs, high mortality)?
|
1) Velogenic newcastle disease
2) Fowl cholera 3) Infectious laryngotracheitis |
|
What is a disease that's reportable in birds?
|
Avian influenza
|
|
What 4 samples should you collect from a bird on necropsy if you suspect avian influenza?
|
1) Serum
2) Tracheal and cloacal swabs 3) Fresh lung, spleen 4) Formalin fixed samples of wide variety of tissues |