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29 Cards in this Set

  • Front
  • Back
layers of skin
epidermis - 5 layers
dermis
hypodermis - areolar/adipose subq tissue/smooth muscle
epidermis
2 layers of interest
stratum corneum - outer, dead keratinized cells
stratum germinativum - epithelial cells are produced
dermis
sweat/sebaceous glandd
hair follicles
CT
capillaries, nerves
may extend into epidermis
functions of skin
protect
barrier
sensory
heat regulation
excretion/absorption
production of vitamin D - allows for calcium to be absorbed
body image
severity of burns
BSA
depth
location
age
PMH
cause
superficial thckness
first degree
epidermis only
sunburn - red/swollen, dry, painful
heals within 2-7 days, fluid, cool compress, tylenol
partial thickness
second degree
superfical/deep
epidermis/dermis injured OR totally destroyed
wet, blisters, weeps, edema,
hypertrophic scarring, risk for infection, may graft
full thickness
3rd or 4th degree
epidermis/dermis destroyed
hard, leathery, waxy, brown/yellow/black
no painful - will not regenerate
4th degree = bone/muscle burned
graft/amputate
rule of 9s
each arm 9% (front/back each 4.5%)
each leg 18% (front/back each 9%)
torso 18% front, 18% back
head 9% (front/back each 4.5%)
hand 1% each
lund and browder
factors in age
why BSA
fluid/nutrition needs
4mLxtotal kgx%of body burned
1/2 in first 8 hours
1/2 in second 16 hours
thermal burns
hot or cold
fire, flash, scald
zone of coagulation = dead/damaged tissue
zone of stasis = surronding area
zone of hyperemia
chemical burns
acid vs alkali burns = alkali are harder to neutralize
remove clothing - lavage with water 20-30 minutes
tissue damage up to 72 horus
electrical burns
entry and exit site (exit usually lower)
possible cardiac arrest
possible thermal burn
other traumatic injuries
CV system effects
vasodilation - increased capillary permeabililty
proteins leak - water/electrolytes leak
hypovolemic shock d/t 3rd spacing + thick blood
risk for srythmias
renal system
ATN d/t decreased perfusion + myoglobins
GI system
ileus + ulceration d/t decreased perfusion
curlings ulcers, gastritis, ischemic enterocolitis
neurological system
confusion to coma d/t decreased perfusion and CO
respiratory system - smoke inhalation
CO poisoning- takes place of O2
early/late sx - flue, red skin, dizzy, to syncope, convulsions, cardiac arrest
chemical pneumonitis - inflamed mucosa/edema/hemorrhage/spasm, ARDS d/t decreased surfactant
respiratory system- thermal injuries
upper airway -
edema, occlusion, = scope + airway if suspected
blisters on face/mouth, singed hair, difficulty swallowing
lower airway
metabolic system
hypermetabolism - stress, healing, loss of heat, fever,infection
increased protein needs - loss through wounds, increased breakdown, increased metabolic rate, -nitrogen balance
immune system - sepsis
loss of integument
eschar - necrotitric materials
nutritional deficits - cannot fight infection
suppressed immune response
integumentary system
decreased wound healing
scar formation - hypertrophic
msk system
muscle atrophy
scar contactures
bone deformity
hypertrophic bone formation
emergent phase of burn treatment
initial phase - shock , decreased CO
fluid mobilizes, remobilizes
diuretic stage
acute phase
diuresis until wounds healed, covered with skin grafts
rehab phase
restore fx, psychosocial issues
FVD interventions
2 large bore IV cathetars
foley cath
fluid resucciation
vaseline labs, chest x-ray
Na/K
assess adequate fluid replacement
UO, SG
pulse, BP
aterial pH
sensorium
CVP,PCWP, CO
assess for overload
labs