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32 Cards in this Set
- Front
- Back
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Dieuretic most likely to produce profound diuresis and cardiovascular collapse
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Furosemide
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verapamil and digoxin both
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slow conduction through the AV node and sometimes are used together for ventricular control of a patient in Afib/flutter
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effective for short term and long term prophylaxis of PSVT
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Verapamil because of its ability for slowing AV conduction
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Verapamil is effective for short term and long term prophylaxis of PSVT because
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of its ability to slow AV conduction
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statins exert there effects primarily on
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total Cholesterol and LDL levels
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total Cholesterol and LDL levels
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statins exert there effects primarily on
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colesevalam, colestepol and cholestyramine are
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bile acid binding (anion exchange resisn)
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inhibits peripheral LPL and decreases hepatic extraction of FFAs
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gemfibrozil as well as inhibiting the synthesis of apolipoprotein B a carrier molecule for VLDL
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gemfibrozil inhibits peripheral LPL and decreases hepatic extraction of FFAs as well as
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inhibiting the synthesis of apolipoprotein B a carrier molecule for VLDL
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Azetazolemide does what
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increases tubular bicarb, decreases ammonia excretion and increases potassium excretion
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Azetazolemide does what to K excretion
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increases it
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African Americans dont respond as well to
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ACEs or Beta Blockers like they do Calcium channel and Diuretics
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effects of nitro
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increased HR, decreased afterload and preload. NO CHANGE IN CORONARY BLOOD FLOW, decreased oxygen demand
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dobutamine activates Beta 1 receptors that are coupled to what type of G proteins
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Gs therefore it works by increasing cAMP
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to control ventricular rate in folks with AFIB
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Digoxin
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prinzmetal, stable angina, unstable angina with preserved ventricular fxn, HTN, PSVT (verapamil too) are all indications of
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diltiazam
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hypercalcemia hyperglycemia and hyperlipidemia are side effects of what diuretic class
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HCTD
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what cotreatmen is required with minoxidil as opposed to hydralazine
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minoxidil is a potent vasodilator that causes fluid retention and reflex tach so it must be administered with a LOOP and ATENOLOL while with hydralazine cotreatment with HCTD and a beta blocker often suffices
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agents like PIMODOL and ACEBUTOL have ISA and have no effect on
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plasma LIPID levels
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these beta blockers decrease blood pressure without a substantial decrease in the resting heart rate, CO or stroke volume
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carvediol/labetelol because they block alpha and beta receptors
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propanolol is techniqically a class II ANti arrhythmic but also has
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quinidine like effects i.e. decreased upslope of depolarization
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effective in reducing afterload preload and remodeling of the heart
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the ace inhibitors
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nefedipine should be avoided or used with caution in patients with CHF
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Nifedipine
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common side effects of adenosine treatment as in when an IV bolus is given for emrgent PSVT
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BRONCHOSPASM, AV BLOCK, flushing, DYSPNEA
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magnesium can treat torsaide de pointes associated with
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magnesium depletion, cardiac glycosides
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what lipid lowering drug can potentiate the effects of warfarin
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gemfibrozil by displacing it from plasma protein binding sites
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Digoxin prolongs the
depresses the supresses or reverses the and shortens the |
PR interval
ST segment T wave qt interval |
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digoxin on QT
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shorter
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digoxin on PR interval
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longer
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digoxin on ST
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depressing
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digoxin on the T wave
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flattened or reversed
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rapid withdrawal from clonidine can result in severe
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reobound HTN
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