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49 Cards in this Set

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palatal cysts in newborns
multiple keratin filled cysts derived from dental lamina; disappear spontaneously
nasolabial cyst
cyst in upper lip, lateral to midline lined by respiratory epithelium
nasopalatine duct cyst
intraosseous cyst derived from epithelial remnants of nasopalatine duct; on radiograph, presents as a heartshaped radiolucency b/w roots of central incisors; can be confused w/ apical periodontal cyst (which are associated w/ dead teeth)
median palatal cyst
fissural cyst from epithelium entrapped furing fusion of palate; much less comon than and can be confused with nasopalatine cyst
epidermoid cyst of skin
derived from hair follicle; common in head/neck region; cyst lined by epidermis epithelium
dermoid cyst
occurs in mouth; derived from remnants of embryonic skin -> considered a "stripped down teratoma" i.e. has tissue derived from different germ layers; cyst lined by epidermis epithelium; cyst wall contains skin appendages
epidermoid cyst of the mouth
a simple form of dermoid cyst; contains no skin appendages
thyroglossal duct cyst
derived from developmental epithelial remnants of thyroglossal duct; soft movable swelling anywhere b/w thyroid gland and foramen cecum at base of tongue; mix of epithelial, thyroid, lymphoid and glandular tissue
oral lymphoepithelial cyst
yellow-white superifical nodule locaed where lymphoid tissue is present in mouth (post lateral tongue, tonsillar pillars, floor of mouth); lined by parakeratotic stratified squamous epithelium suffounded by lymhoid tissue
cervical lymphoepithelial cyst (branchial cleft cyst)
derived from developmental remants of branchial clefts in lateral neck, often along anterior border of SCM; lined by parakeratotic stratified squamous epithelium suffounded by lymhoid tissue; can include respiratory epith
hemihyperplasia
asymmetry at birth; unilateral overgrowth of tongue, teeth and bones; associated w/ increased incidence of wilm's tumor (kidney tumor in children)
progressive hemifacial atrophy
proressive atrophic (degenerative) changes affecting one side of face appearing during childhood
crouzon and apert syndrome
craniosynostosis; frog-like face e.g. hypertelorism (eyes far apart); mandibular prognathism; apert syndrom characterized by syndactyly of hands and feet
treacher collins syndrome
defective ossification -> midfacial and mandibular hypoplasia and low set ears; downward slanting of eyes; colobomas: notch on outer portion of eyelid; cleft palate is common
fluorosis
ingestion of excess fluoride; amelogenin retained in enamel -> hypomineralized enamel
turner's tooth
caries/truama in primary tooth transmits inflammation to succeeding permanent tooth -> localized distubrance in matrix formation, calcification, shape and color
syphilitic hypoplasia
congenital syphils pts; hutchinson's incisor (screwdrive shaped w/ central notch); mulberry molars (lobulated occusal surface)
attrition
tooth-to-tooth contact causing wear; most common on incisal and occusal surfaces
erosion
caused by nonbacterial chemicals e.g. acidic drinks (anterior labial), vomitting (lingual surfaces)
abrasion
caused by nonmasticatory friction e.g. brushing (buccal/labial), toothpicks (interproximal)
internal tooth resorption
rare; begins from pulp chamber and extends outward; pink tooth of mummery: pulp chamber replaces w/ granulation tissue (inflammation)
external tooth resorption
common; begins at surface and extends inward by PDL cells; caused by local trauma, reimplanted avulsed teeth, orthodontics
tetracycline staining
avoid giving tetracycline to pregnant women or kids <9 y/o; minocycline can discolor adult permanent teeth by staining pulp
congenital erythropoetic porphyria
genetic disorder causing increased porphyrin synthesis -> porphyrin deposited in teeth -> red-brown discoloration
hyperbilirubinemia (jaundice)
usu. limited to primary dentition; bilirubin broken down into biliverdin -> yellow-green staining; can be caused by erythroblastosis fetalis (Rh factor incompatibility -> hemolytic anemia) or biliary atresia
ankylosis
fusion of tooth to surrounding bone; possibly caused by periapical inflammation and subsequent bone repair; more common in primary teeth -> affects permanent successor eruption
oligodontia, andontia, hypodontia
oligodontia: missing more than 6 permanent teeth; andontia: missing all permanent teeth; extreme cases, evaluate pts for ectodermal dysplasia
natal tooth
tooth arising in newborn in first 30 days; mandibular incisors most common
germination
incomplete division of a tooth into two
fusion
union of two separated tooth germs; count reveals a missing tooth
concrescence
union of two adjacent teeth by cementum; can be developmental or due to inflammation
talon cusp
on surface of anterior tooth on cingulum -> occusal clearnace problem
dens invainatus
dens en dente; invagination lined by enamel; coronal>radicular (root); predisposes tooth to decay and pulpitis -> prophylactic filling
dens evaginatus
accesory cusp present in central groove; relatively common; attrition/fracture of cusp can expose pulp; most common in premolars
enamel pearl
ectopic enamel droplet in furcation or on root of molars
cervical enamel projection
apical extensions of enamel past normallysmooth CEJ; PDL cant attach enamel -> prone to perio problems
taurodontism
elongated crown and apically placed furcation -> enlarged rectangular pulp chamber
hypercementosis
excessive cementum deposits; generalized is very rare -> when present consider pagets bone disease; can lead to extraction problems
dilaceration
sharp bend in root
amelogenesis imperfecta (AI)
hereditary; mutated AMELX (amelogenin prot) and ENAM (enamelin) genes
hypoplastic AI
reduced enamel thickness
hypomaturation AI
enamel is normal thickness, but not of normal hardness and translucency; radiodensity similar to dentin
hypocalcified AI
enamel is normal thickness but soft; radiodensity is less than dentin
dentinogenesis imperfecta (DI)
distrbance in dentin formation caused by mutation in DSPP gene; opalescence in teeth of both dentition; not good crown candidates; 3 types
3 shield types of DI
type I: OI+DI (mutation in COL1A1 and COL1A2 genes); type II: classical (blubous crowns, cervical constriction, loss of pulp and root canals; type III: bradywine isolate ("shell teeth"; very thin dentin and enlarged pulp; DSPP mutation)
dentin dysplasia
hereditary defect in dentin formation affecting both primary and permanent dentitions; 2 types
radicular (type I) dentin dysplasia
crowns normal color; short roots and total obliteration of pulp canals; can have chevron shaped pulp or pulp stones; root dentin described as stream flowing around boulders; primary teeth wil always have periapical inflammation
coronal (type II) dentin dysplasia
associated w/ DI and also caused by DSPP mutation; primary DD: opalescent color, bulbous crowns w/ cervical constriction, obliteration of pulp (resembles DI); permanent: clinically normal crown, large coronal pulp w/ apical extension ("thistle tube"), almost always get pulp stones
regional odontodysplasia
affects local area; ghost teeth: thinly mineralized enamel and dentin; large pulp, short root w/ open apex; delayed/failed eruption