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49 Cards in this Set
- Front
- Back
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palatal cysts in newborns
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multiple keratin filled cysts derived from dental lamina; disappear spontaneously
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nasolabial cyst
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cyst in upper lip, lateral to midline lined by respiratory epithelium
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nasopalatine duct cyst
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intraosseous cyst derived from epithelial remnants of nasopalatine duct; on radiograph, presents as a heartshaped radiolucency b/w roots of central incisors; can be confused w/ apical periodontal cyst (which are associated w/ dead teeth)
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median palatal cyst
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fissural cyst from epithelium entrapped furing fusion of palate; much less comon than and can be confused with nasopalatine cyst
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epidermoid cyst of skin
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derived from hair follicle; common in head/neck region; cyst lined by epidermis epithelium
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dermoid cyst
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occurs in mouth; derived from remnants of embryonic skin -> considered a "stripped down teratoma" i.e. has tissue derived from different germ layers; cyst lined by epidermis epithelium; cyst wall contains skin appendages
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epidermoid cyst of the mouth
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a simple form of dermoid cyst; contains no skin appendages
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thyroglossal duct cyst
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derived from developmental epithelial remnants of thyroglossal duct; soft movable swelling anywhere b/w thyroid gland and foramen cecum at base of tongue; mix of epithelial, thyroid, lymphoid and glandular tissue
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oral lymphoepithelial cyst
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yellow-white superifical nodule locaed where lymphoid tissue is present in mouth (post lateral tongue, tonsillar pillars, floor of mouth); lined by parakeratotic stratified squamous epithelium suffounded by lymhoid tissue
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cervical lymphoepithelial cyst (branchial cleft cyst)
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derived from developmental remants of branchial clefts in lateral neck, often along anterior border of SCM; lined by parakeratotic stratified squamous epithelium suffounded by lymhoid tissue; can include respiratory epith
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hemihyperplasia
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asymmetry at birth; unilateral overgrowth of tongue, teeth and bones; associated w/ increased incidence of wilm's tumor (kidney tumor in children)
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progressive hemifacial atrophy
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proressive atrophic (degenerative) changes affecting one side of face appearing during childhood
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crouzon and apert syndrome
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craniosynostosis; frog-like face e.g. hypertelorism (eyes far apart); mandibular prognathism; apert syndrom characterized by syndactyly of hands and feet
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treacher collins syndrome
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defective ossification -> midfacial and mandibular hypoplasia and low set ears; downward slanting of eyes; colobomas: notch on outer portion of eyelid; cleft palate is common
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fluorosis
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ingestion of excess fluoride; amelogenin retained in enamel -> hypomineralized enamel
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turner's tooth
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caries/truama in primary tooth transmits inflammation to succeeding permanent tooth -> localized distubrance in matrix formation, calcification, shape and color
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syphilitic hypoplasia
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congenital syphils pts; hutchinson's incisor (screwdrive shaped w/ central notch); mulberry molars (lobulated occusal surface)
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attrition
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tooth-to-tooth contact causing wear; most common on incisal and occusal surfaces
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erosion
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caused by nonbacterial chemicals e.g. acidic drinks (anterior labial), vomitting (lingual surfaces)
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abrasion
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caused by nonmasticatory friction e.g. brushing (buccal/labial), toothpicks (interproximal)
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internal tooth resorption
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rare; begins from pulp chamber and extends outward; pink tooth of mummery: pulp chamber replaces w/ granulation tissue (inflammation)
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external tooth resorption
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common; begins at surface and extends inward by PDL cells; caused by local trauma, reimplanted avulsed teeth, orthodontics
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tetracycline staining
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avoid giving tetracycline to pregnant women or kids <9 y/o; minocycline can discolor adult permanent teeth by staining pulp
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congenital erythropoetic porphyria
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genetic disorder causing increased porphyrin synthesis -> porphyrin deposited in teeth -> red-brown discoloration
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hyperbilirubinemia (jaundice)
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usu. limited to primary dentition; bilirubin broken down into biliverdin -> yellow-green staining; can be caused by erythroblastosis fetalis (Rh factor incompatibility -> hemolytic anemia) or biliary atresia
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ankylosis
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fusion of tooth to surrounding bone; possibly caused by periapical inflammation and subsequent bone repair; more common in primary teeth -> affects permanent successor eruption
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oligodontia, andontia, hypodontia
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oligodontia: missing more than 6 permanent teeth; andontia: missing all permanent teeth; extreme cases, evaluate pts for ectodermal dysplasia
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natal tooth
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tooth arising in newborn in first 30 days; mandibular incisors most common
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germination
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incomplete division of a tooth into two
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fusion
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union of two separated tooth germs; count reveals a missing tooth
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concrescence
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union of two adjacent teeth by cementum; can be developmental or due to inflammation
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talon cusp
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on surface of anterior tooth on cingulum -> occusal clearnace problem
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dens invainatus
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dens en dente; invagination lined by enamel; coronal>radicular (root); predisposes tooth to decay and pulpitis -> prophylactic filling
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dens evaginatus
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accesory cusp present in central groove; relatively common; attrition/fracture of cusp can expose pulp; most common in premolars
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enamel pearl
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ectopic enamel droplet in furcation or on root of molars
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cervical enamel projection
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apical extensions of enamel past normallysmooth CEJ; PDL cant attach enamel -> prone to perio problems
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taurodontism
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elongated crown and apically placed furcation -> enlarged rectangular pulp chamber
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hypercementosis
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excessive cementum deposits; generalized is very rare -> when present consider pagets bone disease; can lead to extraction problems
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dilaceration
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sharp bend in root
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amelogenesis imperfecta (AI)
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hereditary; mutated AMELX (amelogenin prot) and ENAM (enamelin) genes
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hypoplastic AI
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reduced enamel thickness
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hypomaturation AI
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enamel is normal thickness, but not of normal hardness and translucency; radiodensity similar to dentin
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hypocalcified AI
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enamel is normal thickness but soft; radiodensity is less than dentin
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dentinogenesis imperfecta (DI)
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distrbance in dentin formation caused by mutation in DSPP gene; opalescence in teeth of both dentition; not good crown candidates; 3 types
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3 shield types of DI
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type I: OI+DI (mutation in COL1A1 and COL1A2 genes); type II: classical (blubous crowns, cervical constriction, loss of pulp and root canals; type III: bradywine isolate ("shell teeth"; very thin dentin and enlarged pulp; DSPP mutation)
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dentin dysplasia
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hereditary defect in dentin formation affecting both primary and permanent dentitions; 2 types
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radicular (type I) dentin dysplasia
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crowns normal color; short roots and total obliteration of pulp canals; can have chevron shaped pulp or pulp stones; root dentin described as stream flowing around boulders; primary teeth wil always have periapical inflammation
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coronal (type II) dentin dysplasia
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associated w/ DI and also caused by DSPP mutation; primary DD: opalescent color, bulbous crowns w/ cervical constriction, obliteration of pulp (resembles DI); permanent: clinically normal crown, large coronal pulp w/ apical extension ("thistle tube"), almost always get pulp stones
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regional odontodysplasia
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affects local area; ghost teeth: thinly mineralized enamel and dentin; large pulp, short root w/ open apex; delayed/failed eruption
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