- Shuffle
Toggle OnToggle Off
- Alphabetize
Toggle OnToggle Off
- Front First
Toggle OnToggle Off
- Both Sides
Toggle OnToggle Off
Front
How to study your flashcards.
Right/Left arrow keys: Navigate between flashcards.right arrow keyleft arrow key
Up/Down arrow keys: Flip the card between the front and back.down keyup key
H key: Show hint (3rd side).h key
PLAY BUTTON
PLAY BUTTON
12 Cards in this Set
- Front
- Back
|
3 Morphological variants of acute inflammation
|
1) suppurative inflammation
2) fibrinous inflammation 3) serous inflammation |
|
Bacteria that promotes purulent inflammation
|
pyogenic
|
|
Chemical mediators of arterior dilation
|
NO, histamine
|
|
Chemical mediators of neutrophil activation
|
leukotriene, chemokines, bacteria
|
|
Chemical mediators of epithelial activation
|
serotonin, histamin, leukotrienes
|
|
4 cardinal signs of inflammation
|
pain, redness, swelling, heat
|
|
Outcomes of inflammation determined by (3)
|
1) severity of tissue damage
2) stem cells w/ in 3) agent causing it |
|
3 possible outcomes of inflammation
|
1) Resolution
2) Fibrous Healing (scar) 3) chronic inflammation |
|
acute abscess vs. chronic abscess
|
pus vs. pus encapsulated by granulous/ fibrous tissue
|
|
3 cell types in chronic inflammatory infiltrates
|
-lymphocytes, macrophages, plasma cells
-following non-resolution of acute -certain injurious agents |
|
Chronic inflammation signs
|
co-existing tissue destruction
organization/repair |
|
Langhan's giant cells vs. foreign body giant cells
|
L-horseshoe nuclei
G-similar to surrounding macrophages |
