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T3/T4
"Thyroid"

1. Inc BMR
2. Add to blood anything used to make energy ---
so that we can do gluconeogenesis & glycogenesis in liver ---
therefore it stims insulin secretion so liver can uptake glucose

3. Tissue Growth
Cortisol
Adrenal Cortex

1. @ Liver: Gluconeogenesis & Glucogenesis
2. @ TT: Stims PRO Catab & Inhibs PRO Synth
Aldosterone
Adrenal Cortex

1. Na+ & H2O reabsorption to Inc BV
2. K+ Excretion
Catecholamines
Adrenal Medulla

1. Add to blood anything that can be used to make energy
2. Inc CV & Resp. Fnctn
Ghrelin
1. Stim GH
2. Inhib Leptin --> Inc Appetite
GH
Ant Pituitary

1. Linear Growth
2. Preserve Lean Body Mass (PRO In Muscle) by using fats for energy
IGF
Liver

1. Primary: GH-Like Effects
2. Secondary: Insulin-Like Effects
GH
Ant Pituitary

1. Linear Growth
2. Preserve Lean Body Mass (PRO In Muscle) by using fats for energy
What is the 'SYMPATHETIC' response on 'DIGESTIVE' (SECRETION)?

What receptor is involved in this?
INHIBITED
CONTRACTION OF INTESTINAL SPHINCTERS

(ALPHA-1 RECEPTOR)
Insulin
Panc

1. Remove from blood anything used to make energy
Pancreatic Polypeptide
Panc F Cells

1. Add to blood anything that can be used to make energy
2. EXCEPT supresses appetite
Endocrine Adipocyte
1. CTL Food Intake Along w/ stomach
2. CTL metab of substrate distribution along w/ counter-reg hormones
Leptin
1. Dec Appetite
2. Inc Lipolysis
3. Permissive for LH, FSH
PTH
1. Inc Blood Ca++ from bone
2. Dec Blood Pi by kidney excretion
Vit D
Inc Blood Ca++ & Pi by stim intest absorption
Calcitonin
1. Dec Blood Ca++ from Bone
2. Inc Pi reabsorption in kidney
3. Dec Ca++ reabsorption
Adiponectin
1. Glycogenesis
2. Lipolysis
Resistin

***Stim by what to do what?***
1. Inhib Glycogenesis
2. Stim Glycogenolysis

3. Stim by insulin to competitively inhib insulin --> above effects
TNF-a
1. Inhib PPAR-gamma -->Inhib of pre-adipocytes to Adipocytes
MCP-1
Stim Macs to release TNF-a
SOCS

***When does it become overexpressed?****
1. Inhib Leptin & IL-6
2. Inhib Insulin Receptor

***Becomes overexpressed if you have too many cytokines***