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26 Cards in this Set

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primary apnea
- occurs after birth
-rapid resp followed by cessation of resp
-stimulation may be effective
secondary apnea
- o2 levels cont to decrease
- LOC
-stimulation is ineffective
- resusitation is needed
Asphyxia
infants at risk
-comp preg, labor or birth
-narcotics shortly before birth
Transient Tachypnea of the newborn
overview
-retaines lung fluid
-rapid respirations soon after birth
-resolves within a few days
Transient Tachypnea of the newborn

infants at risk
- C sect without labor
- asphyxia
- quick delivery
-maternal analgesia, bleeding and DM
-immaturity
Transient Tachypnea of the newborn
s/s
- rapid resp
-retractions
-nasal flaring
- grunting
- mild cyanosis
-hyperinflation of lungs
Transient Tachypnea of the newborn

therapeutic mangaemtn
- supportive
-o2 admin
iv or gavage feesing while tachy
MAS
most often occurs
- in postterm infants who have decreased amniotic fluid and are prone to cord compression
MAS
s/s
- resp distress
- tachypnea
-cyanosis
-retactions
-nasal flaring
-grunting
-rales
- rhonci
-barrelchest form hyperinflation
_CXR shows atelectisis, consolidation and hyperexpansion
MAS
therapuetic manaagmetn
- amnioinfusion with sterile saline
-suctioning at birth of head
- endo intubation?
MAS
Nursing considerations
- notifiy HCP ASAP if mecomium in AF is observed
-monitor infant foe s/s of infection
Persistant Pulmonary HTN
def
- condition in which the vascular resitance of the lungs does not decrease after birth and circulation is impaired
Persistant Pulmonary HTN
causes
-maternal use of NSAIDS or ASA
- asphyxia
-MAS
sepsi
polycythemia
- RDS
Persistant Pulmonary HTN
s/s
- within 24-48 hrs
- tachypnea
- resp distress
- progressive cyanosis
-
Persistant Pulmonary HTN
therapeutic management
- tx of the underlying cause
- relieve pulm vasoconstriction
-sedation-
high frequecy ventilation
-ECMO
-decrease stimuli
Hyperbilirubinemia
pathologic
causes
-incompatilibility between blood of mother and NB
-Rh incompatibility
-infection
-hypothyroidism
-polycythemia
-dm mothers
What level for bili to be visible in the NB face?
5
Hyperbilirubinemia
therapeutic management
-prevention of kernicterus
Hyperbilirubinemia
phototherapy
side effects
-frequent, loose, green stools
-tanning
-graying brown color of the skin and urine
-macular skin rash
-temp lactose intolerance
Exchange transfusions
removes sensitized rbc's, anibodies, unconjugated bilirubin and corrects severe anemia
Bili encephalopathy
s/s
- lethargy
-poor muscle tone, high pitched cry
-absent moro reflex
-seizures
Sepsis
signs in NB
-temp instability
-rash
-tachypnea
-apnea
-resp distress
-color changes
-tachycardia
-hypotension
-decreased peripeheral perfusion
-decreased oral intake
-vomitting/diarrhea
-gastric residuals that are more than half of previous reading
-abd distension
-glucose instability
-decreased muscle tone
-lethargy,
irritability
-bulging fontenel
-jaundice
-hemorrhage
-anemia
-enlarged liver/spleen
-resp failure
-shock
-seizures
IDM
assesment for complications
-congenital anomilies
-trauma
-resp problems
-hypoglycemia
-
Intrauterine drug use
-s/s
-irritability
-jiterriness
-muscular rigisity
-restless
-exaggerated moro
-high pitched cry
-difficult to console
-uncoordinated suck and sawlloe
-frequent reurg or diarrhea
-poor slleong
-yawning/sneezing
-seizures
-sweating
Polycythemia
def
HCT >65
polycythemia cause
-poor intrauterine O2
-post maturity
-maternal HTN
-maternal DM
-delayed cor clamping
-transfusion from one twin to the other