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41 Cards in this Set
- Front
- Back
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alcoholic liver disease
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disease resulting form excessive alcohol ingestion, characterized by fatty liver, hepatitis, cirrhosis
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aromatic amino acids
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aa phenylalanine, tryptophan, tyrosine
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ascites
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accumulation of fluid, serum protein, electrolytes within the peritoneal cavity caused by increased pressure from portal hypertension and decreased production of albumin
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bile
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thick, viscous fluid secreted from liver, stored in the gallbladder, and released into the duodenum when fatty foods enter the duodenum; emulsified fats in intestine and forms cmpds w FA to facilitate their absorption
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BCAA
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a valine, isoleucine, leucine
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alcoholic liver disease
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disease resulting form excessive alcohol ingestion, characterized by fatty liver, hepatitis, cirrhosis
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aromatic amino acids
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aa phenylalanine, tryptophan, tyrosine
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ascites
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accumulation of fluid, serum protein, electrolytes within the peritoneal cavity caused by increased pressure from portal hypertension and decreased production of albumin
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bile
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thick, viscous fluid secreted from liver, stored in the gallbladder, and released into the duodenum when fatty foods enter the duodenum; emulsified fats in intestine and forms cmpds w FA to facilitate their absorption
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BCAA
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aa valine, isoleucine, leucine
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cholangitis
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inflammation in bile ducts; may be acute or sclerosing
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cholecystectomy
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removal of gall bladder
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cholecystitis
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inflammation of gall bladder
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choledocholithiasis
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presence of gallstones in common bile duct
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cholelithiasis
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presence or formation of gallstones
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cholestasis
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suppression of biliary flow
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cirrhosis
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chronic liver disease caused by diffuse necrosis and regeneration, leading to an increase in fibrous tissue formation disrupting the normal liver structure
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fasting hypoglycemia
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low blood glucose caused by decreased availability of glucose from glycogen as result of depressed liver function
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fatty liver
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condition (hepatic steatosis) characterized by the accumulation of excess fat in liver commonly caused by alcohol abuse but also associated with obesity, starvation, intestinal bypass, parenteral alimentation, insulin resistance
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fulminant liver disease
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absence of preexhisting liver disease and development of liver disease with hepatic encephalopthy within 2 mos of onset of illness
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hepatic encephalopathy
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clinical syndrome characterized by impaired mentation, neuromuscular disturbances, and altered consciousness; four stages for progression
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hepatic failure
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conditionin which liver fxn is dimished to 25% or less
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hepatic osteodystrophy
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complication of chronic liver disease in which bone mass declines
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hepatic steatosis
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fatty liver
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hepatitis
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widespread inflammation of liver; usually viral in origin
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hepatorenal syndrome
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functional renal failure w/o anatomic or histopathologic renal changes; assoc w cirrhosis and ascites or with obstructive jaundice
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jaundice
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syndrome characterized by hyperbilirubinemia and deposition of bile pigment, resulting in yellowing of skin, mucous membranes and sclera
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kayser-fleischer ring
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greenish yellow pigmented ring encircling the cornea just within the corneoscleral margin; formed by copper deposits in Descement's membrane of the cornea; occurs in patients with Wilson's disease
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kuipffer cells
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fixed phagocytes in sinusoids of liver
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nonalcoholic steatohepatitis
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intermediate stage in fatty liver disease characterized by accumulation of fat droplets in the hepatocytes and presence of fibrous tissue and acute and chronic inflammatory cells
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pancreatoduodenectomy
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(whipple procedure)
excision of head of pancreas along with encircling loop of duodenum; may include partial gastrectomy |
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pancreatitis
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inflammation of pancreas caused by autodigestion of pancreatic tissue by its own enzymes
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paracentesis
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procedure during which fluid from the abdomen is removed through needle
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portal hypertension
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normally increased blood pressure in portal venous system due to the obstruction of blood flow thorugh liver
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portal systemic encephalopathy
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hepatic encephalopathy
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primary biliary cirrhosis
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immune mediated chronic cirrhosis of liver caused by obstruction or infection of small and intermediate-size intrahepatic bile ducts; the extrahepatic biliary tree and larger intrahepatic ducts are normal; 90% of patients are women
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secondary biliary cirrhosis
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liver damage that results from bile backup as a result of gall bladder disease
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steatorrhea
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presence of excess fat in stool
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varicies
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low pressure veins that become distended from increased pressure; most commonly developed in the lower esophagus and upper stomach
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Wernickle's encephalopathy
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conditiion of damage to the CNS from thiamin deficiency; common in alcoholism
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Wilson's Disease
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autosomal-recessive disorder of copper metabolism in which excessive accumulation of copper occurs in liver, CNS and kidney
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