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16 Cards in this Set

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  • Back
is IIb/3a receptor blocker a standard treatment for STEMIs?

what about for non-STEMIs
no, not for STEMIs (fibrinolytics are standard therapy)

yes, for non-STEMIs they are useful to prevent fibrin production and "red clot" formation
which is better for a AMI pt: stenting or fibrinolytic therapy?
stenting - if you can get pt to cath lab within 60-90 minutes, you'll get better prognosis
pt presents with worsened chest pain and SOB after 10 minutes of walking within the past two months. these sx's disappear, however, after resting for 2 minutes. there has not been a worsening of chest pain in the past two months. Dx?

A. Unstable angina
B. Acute coronary syndrome
C. Stable exertional angina
D. Printzmetal’s angina
E. Heart failure due to obesity
stable exertional angina
what is the most important anti-anginal effect of nitrates?
reducing demand (by reducing venous return, lowering preload and LV pressure, decreasing wall stress and subsequent MVO2)
Unlike nitrates, beta blockers:
a. reduce heart rates
b. block AV conduction (prolong PR interval)
c. dilate coronary arteries directly
d. A and B
D
how do you calculate total cholesterol?
TC = LDL + HDL + VLDL

note: VLDL = TG/5
Lipid Profile:
T-Cholesterol: 260 mg/dl
Triglycerides: 180 mg/dl
HDL-C: 40 mg/dl

LDL?
VLDL = triglycerides/5=36 mg/dl
Total cholesterol = ldl + hdl + vldl
LDL = 260-36-40=184 mg
In terms of CC blockers, what do they do?

what's the difference b/w non-DHP and DHP?
Ca blockers increase supply and reduce demands. Remember that non-DHP blockers DO decrease HR (heart rate lowering Ca blockers). Any DHP will have a “-pine” at the end.
class of drugs that end with "-pril"
ACE inhibitors
class of drugs that end with "-sartan"
angiotensin receptor blockers
which is safer: DHP or non-DHP? why?
DHP’s are safer than non-DHP Ca blockers b/c they don’t reduce HR, block AV conduction or decrease contractility.
which test would you recommend for a pt with possible stable exertional angina?

A. Coronary angiography
B. Treadmill exercise ECG test
C. Echocardiogram
D. Ambulatory ECG Holter Monitor
B
50 y/o AA man, chronic heavy smoker, presents with episodes of chest pain lasting for 15 minutes and resolving on their own. recently, pain has started to occur at rest and even woken him up at night. Dx?
A. Unstable angina
B. Subacute myocardial infarction
C. Stable exertional angina
D. Printzmetal’s angina
E. Panic or anxiety attacks
A. pt has pain that goes away within 15 minutes. it is starting to get worse, so need to be on look out for AMI.
T or F. Smoking, being male, and having a FH of premature CAD, drinking , are all coronary risk factors.
false - drinking is not a risk factor
42 yo AA woman presents with 2 hour history of sudden crushing retrosternal chest pain radiating to neck and jaw. associated with diaphoresis, nausea and dizziness.

ECG showed ST- segment elevation in V1-V4. Occasional ventricular premature beats were noted.

Dx?


A. Acute anteroseptal anterior MI
B. Acute transmural ischemia
C. Acute pericarditis
D. Acute coronary syndrome
E. Acute anterolateral myocardial infarction
A.
which of the following drugs would you use first in a pt like this?
A. Thrombolytic drug
B. Aspirin
C. SL nitroglycerin
D. ACEI
E. Heparin
B.