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20 Cards in this Set

  • Front
  • Back
Beta Blockers
Atenolol
metoprolol
cardivelol
propanolol
Ca# Blockers
Felodipine (plendil)
Amlodipin (norvadc)
Istadipine (dynacirc)
Netepine (procardia)
Ace inhibitors
captopril
fosinopril
lilsinopril
ramipril
Alpha blockers
methyldopa
clonidine
Diuretics
Lasix (flurosemide)
Bumex (bumetamide)
HCTZ (hydrochorothiazide)
K sparring - SAT
Risk factors for HPT
focused on global risk.
dyslipidemia, diabetes, smoking, obesity
Arterial disease
is an endothelial disfunction (inflamitory). penitration of lipids and inflamitory cells.
Foam Cells
monocytes that have fagocytised ldl or vldl and become macrophages.
oral contraceptives effect BP
by changing the metabolism of VLDL & LDL
stress effects BP by
increasing, cortisol, epi, and NE,
NE & EPI are released from and effect BP
chromatin cells in the adrenal medulla in response to sympathetic stimuli. The cause an > in BP by vasoconstriction and > heart rate and contractility
parasympathetic neurons intervate the heart
through the vegas nerve, and serve to slow the heart with ACH
Sympathetis neurons intervate the heart
through the cardiac plexis in the aorta, and > BP by increasing rate and contractility through the stimlation of thr SA node
Diabetic metobolic syndrome
waist >40 in (108 cm) in men, and >35 in (88cm) in women. triglycerides > or = to 150, BP > or = to 130/85, HDL < 40 in men and <50 in women, and glucose > or = tp 110
Inflammatory response
swelling, redness, and tissue warmth (vasodilation and > capillary permeability)
histamine, prostaglandins, leukotrieness, bradykinin, platelet-activating factor
IR
tissue damage
lysosomal enzymes and products released from neutrophils, macrophages, and other inflamitory cells.
IR
Chemotaxis
Complement fragments
IR
Pain
Bradykinin
prostaglandins
IR
Fever
Interleukin - 1, Interleukin - 6,
TNF
IR
Leukocytosis
Inerleukin - 1, other cytokines