Reading...
Play button
Play button
Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
19 Cards in this Set
- Front
- Back
|
Identify common infectious agents that can cause vulvitis
|
-HPV -herpes genitalis -gonococcal suppurative infection -syphillis -candidal vulvitis
|
|
|
Identify common infectious agents that can cause vulvitis
|
-HPV -herpes genitalis -gonococcal suppurative infection -syphillis -candidal vulvitis
|
|
|
Identify common infectious agents that can cause vulvitis
|
-HPV -herpes genitalis -gonococcal suppurative infection -syphillis -candidal vulvitis
|
|
|
Describe the presentation of contact dermatitis of the vulva, and distinguish b/t irritant and allergic causes;
|
-well defined erythematous weeping and crusting papules and plaques -irritants = urine, detergents, antiseptics, deodorants, ETOH -allergic = perfume, additives in creams, lotions, and soaps, clothing
|
|
|
Describe lichen sclerosus
|
-atrophic epithelium, usually w/ dermal fibrosis -increased risk of squamous cell carcinoma -appears as smooth, white plaques or papules -when entire vulva is involved-> labia becomes atrophic and stiffened and the vaginal orifice is constricted
|
|
|
Describe lichen simplex chronicus
|
-thickened epithelium, usually with inflammatory infiltrate -presents as a an area of leukoplakia -often present at margins of established CA of the vulva but there is no associated increased predisposition
|
|
|
Condylomata lata
|
-flat, moist, minimally elevated lesions that occur in secondary syphillis -less common
|
|
|
Condylomata acuminata
|
-more common -may be papillary and distinctly elevated or somewhat flat and rugose -usually in multiple sites
|
|
|
Two types of squamous cell carcinoma of the vulva
|
-HPV=present in 75-90% of the cases, younger,smokers,co-existing vaginal or cervical carcinoma,usually poorly differentiated lesions&evolve from vulvar intraepithelial neoplasia-non-HPV=older, well differentiated&unifocal,associated with lichen scelorsus
|
|
|
Two most common causes of vaginitis
|
-Candida albicans -Trichomonous vaginalis
|
|
|
Three most important causes of cervictis
|
-Chlamydia trachomatis -Neisseria gonorrhea -herpes simplex
|
|
|
Anatomy of the squamocolumnar junction and the transformation zone and its importance as the site of cervical cancer
|
-
|
|
|
Describe the CIN grading system for cervical dysplasia
|
-I= mild dysplasia -II=moderate dysplasia -III=severe dysplasia and carcinoma in situ
|
|
|
Describe the sequence of events that may follow from HPV infection, to CIN, to invasive cervical carcinoma
|
-sex_> HPV-> HPV exposure-> low risk HPV (6,11) =condyloma -high risk (16,18)-> CIN-> higher grade CIN-> invasive CA-> metastasis
|
|
|
Identify risk factors for the development of CIN and invasive cervical carcinoma
|
-early age at first intercourse -multiple sexual partners -a male partner w/ multiple previous sexual partners -persistnent infection by "high-risk" papillomaviruses
|
|
|
Briefly describe the cause and clinical presentation of endometritis
|
-inflammation of endometrium -part of a larger spectrum of pelvic inflammatory disease -consequences for the integrity of fallopian tubes, fertility, may be associated retained products of a miscarriage or delivery -acute=N. gonorrhea or C. trachomatis ?
|
|
|
Cause and clinical presentation of adenomyosis
|
-growth of the basal layer of the endometrium to the myometrium -uterine wall is enlarged -may produce menorrhagia, dysmenorrhea, pelvic pain prior to menstarion -no cyclical bleeding
|
|
|
Cause and clinical presentation of endometriosis
|
-endometrial glands and stroma outside the uterus and may involve the pelvic or abdominal peritoneumm and sometimes distant lymph nodes -may present w/ a pelvic mass filled w/ degenerating blood (chocolate mass) -three theories of origin
|
|
|
Endometriosis and the regurgitation theory
|
-menstral backflow through the fallopian tubes w/ subsequent implantation of lesion
|
