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12 Cards in this Set
- Front
- Back
- 3rd side (hint)
PVT
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>180bpm, very wide QRS complex
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originates in VENTRICLE +PULSELESS
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Ventricular Fibrillation
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uncoordinated ventricular contractions
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HYPOXIA primary cause, can lead to ischemia & asystole
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Pulseless Electrical Activity PEA
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ALWAYS CHECK FOR PULSE 1ST!
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HYPOVOLEMIA primary cause, treat like asystole,
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ASYSTOLE
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true state of NO Electrical Activity
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1st ° AV Block
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PR interval > 0.20 sec
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of no consequence UNLESS MI or electrolyte imbalance
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2nd ° Heart Block type 1
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Also called Mobitz 1, PR interval prolonged till atrial impulse blocked, thus, no QRS impulse
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Atrial rhythm will be REGULAR
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2nd ° Heart Block type 2
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Mobitz II, almost always disease of distal/ventricular conduction
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non conducted P waves, thus no QRS; no PR prolongation, wide QRS complex CAN LEAD TO COMPLETE BLOCK, txt= transcutaneous/transvenous pacing, or *Atropine
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Complete Heart Block
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3rd °, impulse generates in SA node, not conducted to ventricles
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most common causes infarction & ischemia; txt= transcutaneous pacing; s&s= bradycardia, hypotension, hemodynamic instability
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Supraventricular Tachycardia SVT
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drop in cardiac output, HR >150 bpm
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unstable, txt= cardioversion S&S= SOB, Palpitations, angina, dizziness, LOS, parasthesia, hyperventilation
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Atrial Flutter
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atrial contractions 240-350 bpm due to electrical activity in SA/AV node loop
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S&S= palpitations, SOB,dizziness, nausea, impending doom, Peripheral edema, activity intolerance EARLY CARDIOVERSION
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A FIB
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SA node overwhelmed by atrial impulses, leads to irregular impulses to ventricles
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no P -waves before QRS, HR is IRREGULAR, same S&S as A flutter
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Other Tachycardias
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monomorphic will deteriorate to V-Fib; polymorphic originates in ventricles rather than normal rhythm (from atria)
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