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40 Cards in this Set

  • Front
  • Back

the cardiac muscle is capable of?

autorhythmicity

phase 2 of the cardiac action potential, when the calcium channels remain open and potassium channels are closed, is called the?

plateau phase

which of the following is true of the cardiac action potential?

the cardiac action potential is longer than the skeletal muscle action potential

the main anatomical difference between between the frog heart and the human heart is that the frog heart has

a single, fused ventricle

the amplitude of the ventricular systole did not change with the more frequent stimulation because?

new contraction could not begin until the relaxation period

which of the following do you think contribute to the inability of cardiac muscle to be tetanized?

a long refractory period of the cardiac action potential

given the function of the heart, why is it important that cardiac muscle cannot reach tetanus?

the ventricles must contract and relax fully with each beat to pump blood

an extrasystole corresponds to?

an extra ventricular contraction

the effect of the parasympathetic NS on the heart is to?

decrease heart rate

the branch of the autonomic NS that dominates during exercise is?

sympathetic branch

parasympathetic stimulation reaches the heart through

vague nerves, which are cranial nerves

the usual pacemaker of the heart

is the SA node

extreme vagus nerve stimulation affects the heart by

stopping the heart completely

vagal escape probably involves

sympathetic reflexes

research shows that in the absence of neural and hormonal influences the sa node generates action potentials at a frequency of 100 times per minute. however the resting heart rate is 70 beats per minute which suggests that

the parasympathetic NS has more control over the heart rate

the sa node in the human heart is located

in the right atrium

organisms that usually maintain the same internal body temp in spite of environmental temp changes are

homeothermic

the general name for the process that maintains the internal body temperature in humans is

homeostasis

the electrolytes in a ringers solution are required to

provide for autorhythmicity

an internal body temp that is above normal range is

hyperthermic

the 5 degrees solution, the frog heart

would be slower than baseline

in the 32 degrees solution the frogs heart would

beat faster than baseline

if the human heart we’re experiencing hypothermia, what affect would it have on heart rate?

decrease heart rate

without the ringers solution

spontaneous cardiac action potentials would not occur

the parasympathetic NS releases ___________ to affect heart rate

acetylcholine

a cholinergic drug that worked the same as acetylcholine would

be an agonist and decrease heart rate

norepinephrine affects the heart rate by

increasing the rate of depolarization and increasing frequency of action potentials

the _________ receptor binds to norepinephrine and epinephrine

beta 1 adrenergic

pilocarpine decreased the heart rate. typical cholinergic agonists, it

decrease the frequency of action potentials

the effect of atropine was to

mimic the sympathetic NS

the modifiers tested that decrease the heart rate were

digitalis and pilocarpine

to increase the heart rate the best choices would be

epinephrine and atropine

which organelle in cardiac muscle stores calcium

sarcoplasmic reticulum

verapamil is a calcium channel blocker. it’s effects would be described as

negative chronotropic and negative inotropic

when the cardiac muscle cell is at rest, where is most of the potassium found?

in cytosol

resting cardiac muscle are most permeable to

potassium

the addition of most ions resulted in

erratic heart rate

the effect of potassium on the heart is

negative chronotropic and inotropic

the ion that had the most pronounced effect on the heart rate was

potassium

ectopic pacemakers can be caused by excessive leakage of potassium into cardiac cells, resulting in pacemakers appearing in abnormal locations in the heart muscle. this hyperkalemia decreases the resting potential of the cardiac muscle cell. what effect do you think this would have on the force of contraction

decrease, negative inotropic