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36 Cards in this Set

  • Front
  • Back
High pitch, Loud amplitude, inspiration
Bronchial (Tracheal)
inspiration=expiration; moderate pitch; moderate amplitude; mixed sounds; heard over major bronchi where there are less alveoli (i.e.; between scapulae, around upper sternum;
Bronchovesicular
inspiration>expiration; low pitch; soft amplitude; rustling sound; heard over the peripheral lung fields where air flows through smaller bronchioles and alveoli
Vesicular
difficulty breathing when supine; state number of pillows needed to achieve comfort
Orthopnea
awakening from sleep with SOB and needing to be upright to achieve comfort
Paroxysmal nocturnal dyspnea
discontinuous, high-pitched, short crackling, popping sounds heard during inspiration; not cleared by coughing
Fine crackles
Clinical Example - Occurs with restrictive disease; pneumonia, heart failure, interstitial fibrosis
Late inspiratory crackles
Clinical Example - Occurs with chronic bronchitis, asthma and emphysema
Early inspiratory crackles
Clinical example- Fine crackles appear with change from sitting to supine position
Posturally induced crackles
Loud, low-pitched, bubbling and gurgling sounds; start in early inspiration; may be present in expiration; (sounds like opening a velcro fastener)
Coarse crackles
Clinical Example- Occurs in pulmonary edema, pneumonia, pulmonary fibrosis, terminally ill with depressed cough
Coarse crackles
sounds like fine crackles; do not last; not pathologic; disappear after first few breaths
Atelectatic crackles
Clinical example- In aging adults, in bedridden persons, or in persons just aroused from sleep
Atelectatic crackles
superficial sound; coarse and low pitched; grating quality; sounds just like crackles but close to the ear; two pieces of leather being rubbed together
Pleural friction rub
Clinical example- Pleuritis, accompanied by pain with breathing
Pleural Friction rub
These are discrete, crackling sounds
Discontinuous Sounds
These are connected, musical sounds
Continuous Sounds
High-pitched, musical speaking sounds that sound polyphonic; predominate in expiration but may occur in both expiration and inspiration
High-pitched (sibilant) Wheeze
Clinical example- diffuse airway obstruction from acute asthma or chronic emphysema
High-pitched (sibilant) Wheeze
Low-pitched; monophonic, single note, musical snoring, moaning sounds; heard throughout the cycle; more prominent in expiration; may clear somewhat by coughing
Low-pitched (sonorous rhonchi) Wheeze
Clinical example- Bronchitis, single bronchus obstruction from airway tumor
Low-pitched (sonorous rhonchi) Wheeze
high-pitched, monophonic, inspiratory, crowing sound; louder in neck than chest wall
Stridor
Clinical example- occurs in croup, acute epiglottitis in children, and foreign inhalation; may be life threatening
Stridor
collapsed shrunken section of alveoli or entire lung; results in 1) airway obstruction, alveolar air beyond is gradually absorbed by pulmonary capillaries, alveolar walls cave in; 2) compression on the lung; and 3) lack of surfactant (hyaline membrane disease)
Atelectasis (collapse)
pump failure with increasing pressure of cardiac overload causing pulmonary congestion or increased amount of blood present in pulmonary capillaries; dependent air sacs are deflated; pulmonary capillaries are engorged; bronchial mucosa may be swollen
Heart failure

Inspection: increased respiratory rate, SOB on exertion, orthopnea, paroxysmal nocturnal dyspnea, nocturia, ankle edema, pallor;


Palpation: moist, clammy skin; fremitus normal; Percussion: resonant;


Auscultation: normal vesicular; hear sounds include S3 gallop;


Adventitious sounds: crackles at lung bases.

Signs of heart failure
RBC’s and WBC’s pass from blood to alveoli, alveoli progressively fill with bacteria, solid cellular debris, fluid and blood cells, decreases surface area of respiratory membrane; causes hypoxemia;
Lobar pneumonia
Excessive mucus secretion; inflammation of bronchi with partial obstruction by secretions or constrictions; acute or chronic;
Bronchitis
destruction of pulmonary connective tissue (elastin, collagen); permanent enlargement of air sacs; increased airway resistance, especially on expiration producing hyperinflated lung and an increase in lung volume (cigarette smoking 80-90% cases)
Emphysema
allergic hypersensitivity to inhaled allergens, irritants, microbes, stress or exercise, producing bronchospasm and inflammation, edema in bronchiole walls, secretion of viscous mucus to airways; symptoms include: wheezing, dyspnea, chest tightness;
Asthma
collection of excess fluid in intrapleural space with compression of overlying lung tissue; may contain watery, capillary fluid (transudative), protein (exudative), purulent matter (empyemic), blood (hemothorax), or milky lymphatic fluid (chylothorax);
Pleural effusion (fluid) or thickening
free air in pleural space causing partial or complete lung collapse; air neutralized the usual negative pressure that is present; can be 1) spontaneous (air enters space through puncture lung wall); 2) traumatic (air enters through opening or injury in chest wall); and 3) tension (trapped air in space increases, compressing lung);
Pneumothorax
virulent form of pneumonia; protozoal infection associated with AIDS; common in US but harmless to most people, except immunocompromised; cysts form in alveolar spaces, thickening their walls;
Pneumonia
inhalation of tubercle bacilli into alveolar wall; initial acute inflammatory response, then scar tissue forms, lesions calcify, reactivation of previously healed lesions, bacilli multiply; subjective: initially asymptomatic; weight loss; anorexia; easily fatigued; low grade afternoon fever; night sweats;
Tuberculosis
undissolved material originating in the legs/pelvis, detach and travel through venous system returning blood to R heart, which occludes pulmonary vessels; 99% arise from DVT; pulmonary occlusion results in ischemia of lung tissue, increased pulmonary artery pressure, decreased cardiac output and hypoxia; subjective: chest pain, worse on deep inspiration, dyspnea;
Pulmonary embolism
acute pulmonary insult damages alveolar capillary membrane leading to increased permeability of pulmonary capillaries and alveolar epithelium and pulmonary edema;
Acute respiratory distress syndrome (ARDS)